Building muscle on a caloric deficit?

R

Razien99

New Member
Ok so i just came across this small article about an experiment performed on rats showing that their muscles could grow regardless of the diets the rats were on.

"The experiment was performed to determine the effects of nutrition on muscle building. Procedures were performed on rats to isolate certain leg muscles, and the rats were forced to exercise those muscles. Groups of rats were fed different diets. Some were fed only water. Some were even altered to eliminate insulin production or growth hormone production. The results showed that the exercised muscles grew regardless of starvation diets or adverse conditions. The exercised muscles grew even though the total weight of the animals dropped by around 30 percent."

"If you are consuming less calories than your body needs each day and building muscle, your body will have to find another source for those calories. That source is FAT."

Just wondering how accurate this is out of curiosity. Can fat be converted back into amino acids?
Surely based on that, you can continue to build muscle whilst cutting. ?
 
Not one to be critical, but doing a search for that name in that Journal provides nothing for the reference he provided.

THere is one from Goldberg that year in that journal, listed below

Med Sci Sports. 1975 Fall;7(3):185-98. Related Articles, Links

Mechanism of work-induced hypertrophy of skeletal muscle.

Goldberg AL, Etlinger JD, Goldspink DF, Jablecki C.

Skeletal muscle can undergo rapid growth in response to a sudden increase in work load. For example, the rat soleus muscle increases in weight by 40% within six days after the tendon of the synergistic gastrocnemius is sectioned. Such growth of the overworked muscle involves an enlargement of muscle fibers and occasional longitudinal splitting. Hypertrophy leads to greater maximal tension development, although decreased contraction time and reduced contractility have also been reported. Unlike normal developmental growth, work-induced hypertrophy can be induced in hypophysectomized or diabetic animals. This process thus appears independent of growth hormone and insulin as well as testosterone and thyroid hormones. Hypertrophy of the soleus can also be induced in fasting animals, in which there is a generalized muscle wasting. Thus muscular activity takes precedence over endocrine influences on muscle size. The increase in muscle weight reflects an increase in protein, especially sarcoplasmic protein, and results from greater protein synthesis and reduced protein breakdown. Within several hours after operation, the hypertrophying soleus shows more rapid uptake of certain amino acids and synthesis of phosphatidyl-inositol. By 8 hours, protein synthesis is enhanced. RNA synthesis also increases, and hypertrophy can be prevented with actinomycin D. Nuclear DNA synthesis also increases on the second day after operation and leads to a greater DNA content. The significance of the increased RNA and DNA synthesis is not clear, since most of it occurs in interstitial and satellite cells. The proliferation of the non-muscle cells seems linked to the growth of the muscle fibers; in addition, factors causing muscle atrophy (e.g. denervation) decrease DNA synthesis by such cells. In order to define more precisely the early events in hypertrophy, the effects of contractile activity were studied in rat muscles in vitro. Electrical stimulation enhanced active transport of certain amino acids within an hour, and the magnitude of this effect depended on the amount of contractile activity. Stimulation or passive stretch of the soleus or diaphragm also retarded protein degradation. Presumably these effects of mechanical activity contribute to the changes occuring during hypertrophy in vivo. However, under the same conditions, or even after more prolonged stimulation, no change in rates of protein synthesis was detected. These findings with passive tension in vitro are particularly interesting, since passive stretch has been reported to retard atrophy or to induce hypertrophy of denervated muscle in vivo. It is suggested that increased tension development (either passive or active) is the critical event in initiating compensatory growth.

So its basically the reference he is talking about, but if he cannot get a reference correct, what does that rate to everything else.
Hypertrophy of one muscle while others waste is indeed possible, but thats not how we choose to function. Dealing with a whole body is somewhat different to what they are doing.

Losing fat and gaining muscle at hte same time is extremely possible, especially for fatties, but as you get leaner it will be harder and harder to achieve.
 
Yeah thanks for clearing that up, thats something else ive always been curious about. When some people start working out they get beginners gains where they lose fat & build muscle at the same time, is that related to this in anyway?
 
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