I tend to agee with Cliner9er here, one study doesn't suggest that oral supplementation doesn't cause an additive effect to LBM.
There is a reiview that states this exact idea.
J Orthop Sports Phys Ther. 2003 Oct;33(10):615-21. Related Articles, Links
Creatine supplementation and athletic performance.
Racette SB.
Nutritional supplements and other ergogenic aids have gained widespread use among professional, amateur, recreational, and student athletes for their potential to enhance athletic performance and provide a competitive edge. Creatine monohydrate is one of the more commonly used and potentially beneficial supplements that currently is viewed to be safe. Supplementation with oral creatine augments skeletal muscle creatine concentrations in most individuals, which has been shown to promote gains in lean body mass when used in conjunction with resistance training, to enhance power and strength, and to improve performance in intense exercise, especially during repeated bouts........ Variability in research study designs and small sample sizes have left many questions unanswered regarding the safety and efficacy of chronic supplementation. This is an active area of clinical investigation and the results of ongoing and future research should guide the appropriate use of creatine to enhance athletic performance among athletes of all ages.
Also there is direct evidence that some respond to Creatine and some do not.
Acute creatine monohydrate supplementation: a descriptive physiological profile of responders vs. nonresponders.
Syrotuik DG, Bell GJ.
The purpose of this study was to describe the physiological profile of responders (>20 mmol.kg(-1) dry weight [dw] increase in total intramuscular creatine monohydrate [Cr] + phosphorylated creatine [PCr]) versus nonresponders (<10 mmol.kg(-1) dw increase) to a 5-day Cr load (0.3 g.kg(-1).d(-1)) in 11 healthy men (mean age = 22.7 years). Pre-post 5-day cellular measures included total resting Cr content (Cr + PCr), fiber type composition, and fiber type cross-sectional area (CSA) determined from muscle biopsies of the vastus lateralis. Body mass, daily dietary intake, 24-hour urine outputs, urinary Cr and creatinine (CrN), and strength performance measures (1 repetition maximum [1RM] bench and leg press) were also assessed before and after the 5-day loading period. Results indicated that there were 3 levels of response to the 5-day supplementation: responders ®, quasi responders (QR), and nonresponders (NR) with mean changes in resting Cr + PCr of 29.5 mmol.kg(-1) dw (n = 3), 14.9 mmol.kg(-1) dw (n = 5), and 5.1 mmol.kg(-1) dw (n = 3), respectively. The results support a person-by-treatment interaction to acute Cr supplementation with R possessing a biological profile of lowest initial levels of Cr + PCr, greatest percentage of type II fibers, and greatest preload muscle fiber CSA and fat-free mass. Responders also showed improvement in 1RM leg press scores following the 5-day loading period. NR had higher preload levels of Cr + PCr, less type II muscle fibers, small preload muscle CSA, and lower fat-free mass and displayed no improvements in 1RM strength scores. The results suggest that to be considered a responder to acute oral supplementation, a favorable preexisting biological profile may determine the final extent to which an individual responds to supplementation. Physiologic profiles of nonresponders appear to be different and may limit their ability to uptake Cr. This may help partially explain the reported equivocal performance findings in the Cr supplementation literature.
