I just came across something at misc.fitness.weights and thought it may help our HIIT quite a bit for losing fat. I'll post the whole post plus the url. Enjoy.
I am interested in your thoughts on the following section of an
article by Christian Finn. The abstract he bases his comments on is
at the end. Thanks.--James.
Short or long intervals?
Research published in the European Journal of Applied Physiology
compared the fat-burning effects of two different interval training
workouts, both lasting 40 minutes [2].
The first consisted of short intervals lasting 6 seconds, with
9-second rest periods. The second workout involved long intervals
lasting 24 seconds, with 36-second rest periods.
It's important to note that the treadmill speed was identical during
both the short and long interval workouts.
Moreover, the ratio between work and recovery bouts was also the same,
meaning that the total amount of time spent running on the treadmill
(16 minutes) was also identical.
Despite the fact that exercise intensity and duration were kept
constant during both trials, there were large differences in fat
oxidation.
In fact, the number of fat calories burned was approximately 3 times
LOWER during the long (24 seconds) interval workout.
To understand why the short intervals were so much more effective at
increasing fat oxidation, it's important to understand a little more
about a substance called myoglobin (pronounced my-o-globe-in).
What is myoglobin?
Myoglobin is a large protein that binds to oxygen inside your muscle
cells [3]. Think of it a little like a "reserve" oxygen supply.
Per Olof Astrand, whose Textbook of Work Physiology is required
reading for many exercise science students, first proposed a role for
myoglobin as an oxygen store during interval exercise way back in the
1960's [1].
According to Astrand, myoglobin is repeatedly used and reloaded during
the work and recovery phases of interval exercise.
However, as the duration of the work period's increase, myoglobin
stores are reduced.
Your body needs more oxygen to use fat as a fuel (compared to
carbohydrate or protein). When oxygen supplies become limited,
carbohydrate supplies a greater proportion of energy.
Because lactic acid, a by-product of carbohydrate metabolism, "blocks"
fat burning, intervals that continue beyond the point at which
myoglobin loses its supply of oxygen rely to a greater extent on
carbohydrate as a source of energy.
How long do myoglobin stores last?
Myoglobin holds enough oxygen to last for 5-15 seconds [1].
This explains why short, rather than long intervals appear to promote
a greater rise in fat oxidation.
As such, if your goal is to lose fat, then limit your work intervals
to a maximum of 15 seconds.
Studies also show that shorter intervals don't feel as physically
demanding as long intervals -- so you can get better results without
feeling like you're working harder.
How long should my rest intervals last?
This depends on the duration of the work intervals.
The longer the work interval, the more myoglobin gets used up, and the
longer it takes to "reload".
The study we looked at earlier used rest intervals that were 1.5 times
greater than the work intervals (6 seconds work: 9 seconds rest).
Based on these findings, a 15-second work interval would require a
minimum of 22 seconds rest.
If you've never tried interval training, a rest period lasting 45
seconds might be a good place to start.
As your fitness level gradually improves, you'll be able to gradually
reduce your rest time.
Medline Abstract
MA Christmass, B Dawson, and PG Arthur
Effect of work and recovery duration on skeletal muscle oxygenation
and fuel use during sustained intermittent exercise.
Eur J Appl Physiol Occup Physiol, October 1, 1999; 80(5): 436-47.
[Abstract]
--------------------------------------------------------------------------------
Department of Biochemistry, The University of Western Australia,
Nedlands, 6907, Australia. mxmass@cyllene.uwa.edu.au
The purpose of this study was to compare rates of substrate oxidation
in two protocols of intermittent exercise, with identical treadmill
speed and total work duration, to reduce the effect of differences in
factors such as muscle fibre type activation, hormonal responses,
muscle glucose uptake and non-esterified fatty acid (NEFA)
availability on the comparison of substrate utilisation. Subjects (n =
7) completed 40 min of intermittent intense running requiring a
work:recovery ratio of either 6 s:9 s (short-interval exercise, SE) or
24 s:36 s (long-interval exercise, LE), on separate days. Another
experiment compared O(2) availability in the vastus lateralis muscle
across SE (10 min) and LE (10 min) exercise using near-infrared
spectroscopy (RunMan, NIM. Philadelphia, USA). Overall (i.e. work and
recovery) O(2) consumption (VO(2)) and energy expenditure were lower
during LE (P < 0.01, P < 0.05, respectively). Overall exercise
intensity, represented as a proportion of peak aerobic power
(VO2(peak)), was [mean (SEM)] 64.9 (2.7)% VO2(peak) (LE) and 71.4
(2.4)% VO2(peak) (SE). Fat oxidation was three times lower (P < 0.01)
and carbohydrate oxidation 1.3 times higher (P < 0. 01) during LE,
despite the lower overall exercise intensity. Plasma lactate was
constant and was higher throughout exercise in LE [mean (SEM) 5.33
(0.53) mM, LE; 3.28 (0.31) mM, SE; P < 0.001)]. Plasma pyruvate was
higher and glycerol was lower in LE [215 (17) microM, 151 (13) microM,
P < 0.05, pyruvate; 197 (19) microM, 246 (19) microM, P < 0.05,
glycerol]. There was no difference between protocols for plasma NEFA
concentration (n = 4) or plasma noradrenaline and adrenaline. Muscle
oxygenation declined in both protocols (P < 0.001), but the nadir
during LE was lower [52.04 (0. 60)%] compared to SE [61.85 (0.51)%; P
< 0.001]. The decline in muscle oxygenation during work was correlated
with mean lactate concentration (r = 0.68; P < 0.05; n = 12). Lower
levels of fat oxidation occurred concurrent with accelerated
carbohydrate metabolism, increases in lactate and pyruvate and reduced
muscle O(2) availability. These changes were associated with
proportionately longer work and recovery periods, despite identical
treadmill speed and total work duration. The proposal that a metabolic
regulatory factor within the muscle fibre retards fat oxidation under
these conditions is supported by the current findings.
It was found at http://groups.google.com/groups?....weights
I am interested in your thoughts on the following section of an
article by Christian Finn. The abstract he bases his comments on is
at the end. Thanks.--James.
Short or long intervals?
Research published in the European Journal of Applied Physiology
compared the fat-burning effects of two different interval training
workouts, both lasting 40 minutes [2].
The first consisted of short intervals lasting 6 seconds, with
9-second rest periods. The second workout involved long intervals
lasting 24 seconds, with 36-second rest periods.
It's important to note that the treadmill speed was identical during
both the short and long interval workouts.
Moreover, the ratio between work and recovery bouts was also the same,
meaning that the total amount of time spent running on the treadmill
(16 minutes) was also identical.
Despite the fact that exercise intensity and duration were kept
constant during both trials, there were large differences in fat
oxidation.
In fact, the number of fat calories burned was approximately 3 times
LOWER during the long (24 seconds) interval workout.
To understand why the short intervals were so much more effective at
increasing fat oxidation, it's important to understand a little more
about a substance called myoglobin (pronounced my-o-globe-in).
What is myoglobin?
Myoglobin is a large protein that binds to oxygen inside your muscle
cells [3]. Think of it a little like a "reserve" oxygen supply.
Per Olof Astrand, whose Textbook of Work Physiology is required
reading for many exercise science students, first proposed a role for
myoglobin as an oxygen store during interval exercise way back in the
1960's [1].
According to Astrand, myoglobin is repeatedly used and reloaded during
the work and recovery phases of interval exercise.
However, as the duration of the work period's increase, myoglobin
stores are reduced.
Your body needs more oxygen to use fat as a fuel (compared to
carbohydrate or protein). When oxygen supplies become limited,
carbohydrate supplies a greater proportion of energy.
Because lactic acid, a by-product of carbohydrate metabolism, "blocks"
fat burning, intervals that continue beyond the point at which
myoglobin loses its supply of oxygen rely to a greater extent on
carbohydrate as a source of energy.
How long do myoglobin stores last?
Myoglobin holds enough oxygen to last for 5-15 seconds [1].
This explains why short, rather than long intervals appear to promote
a greater rise in fat oxidation.
As such, if your goal is to lose fat, then limit your work intervals
to a maximum of 15 seconds.
Studies also show that shorter intervals don't feel as physically
demanding as long intervals -- so you can get better results without
feeling like you're working harder.
How long should my rest intervals last?
This depends on the duration of the work intervals.
The longer the work interval, the more myoglobin gets used up, and the
longer it takes to "reload".
The study we looked at earlier used rest intervals that were 1.5 times
greater than the work intervals (6 seconds work: 9 seconds rest).
Based on these findings, a 15-second work interval would require a
minimum of 22 seconds rest.
If you've never tried interval training, a rest period lasting 45
seconds might be a good place to start.
As your fitness level gradually improves, you'll be able to gradually
reduce your rest time.
Medline Abstract
MA Christmass, B Dawson, and PG Arthur
Effect of work and recovery duration on skeletal muscle oxygenation
and fuel use during sustained intermittent exercise.
Eur J Appl Physiol Occup Physiol, October 1, 1999; 80(5): 436-47.
[Abstract]
--------------------------------------------------------------------------------
Department of Biochemistry, The University of Western Australia,
Nedlands, 6907, Australia. mxmass@cyllene.uwa.edu.au
The purpose of this study was to compare rates of substrate oxidation
in two protocols of intermittent exercise, with identical treadmill
speed and total work duration, to reduce the effect of differences in
factors such as muscle fibre type activation, hormonal responses,
muscle glucose uptake and non-esterified fatty acid (NEFA)
availability on the comparison of substrate utilisation. Subjects (n =
7) completed 40 min of intermittent intense running requiring a
work:recovery ratio of either 6 s:9 s (short-interval exercise, SE) or
24 s:36 s (long-interval exercise, LE), on separate days. Another
experiment compared O(2) availability in the vastus lateralis muscle
across SE (10 min) and LE (10 min) exercise using near-infrared
spectroscopy (RunMan, NIM. Philadelphia, USA). Overall (i.e. work and
recovery) O(2) consumption (VO(2)) and energy expenditure were lower
during LE (P < 0.01, P < 0.05, respectively). Overall exercise
intensity, represented as a proportion of peak aerobic power
(VO2(peak)), was [mean (SEM)] 64.9 (2.7)% VO2(peak) (LE) and 71.4
(2.4)% VO2(peak) (SE). Fat oxidation was three times lower (P < 0.01)
and carbohydrate oxidation 1.3 times higher (P < 0. 01) during LE,
despite the lower overall exercise intensity. Plasma lactate was
constant and was higher throughout exercise in LE [mean (SEM) 5.33
(0.53) mM, LE; 3.28 (0.31) mM, SE; P < 0.001)]. Plasma pyruvate was
higher and glycerol was lower in LE [215 (17) microM, 151 (13) microM,
P < 0.05, pyruvate; 197 (19) microM, 246 (19) microM, P < 0.05,
glycerol]. There was no difference between protocols for plasma NEFA
concentration (n = 4) or plasma noradrenaline and adrenaline. Muscle
oxygenation declined in both protocols (P < 0.001), but the nadir
during LE was lower [52.04 (0. 60)%] compared to SE [61.85 (0.51)%; P
< 0.001]. The decline in muscle oxygenation during work was correlated
with mean lactate concentration (r = 0.68; P < 0.05; n = 12). Lower
levels of fat oxidation occurred concurrent with accelerated
carbohydrate metabolism, increases in lactate and pyruvate and reduced
muscle O(2) availability. These changes were associated with
proportionately longer work and recovery periods, despite identical
treadmill speed and total work duration. The proposal that a metabolic
regulatory factor within the muscle fibre retards fat oxidation under
these conditions is supported by the current findings.
It was found at http://groups.google.com/groups?....weights