Are you going to fast for a whole day?[b said:Quote[/b] (micmic @ May 02 2003,3:07)]I knew it! This is my new way of dieting!
[b said:Quote[/b] ]Are you going to fast for a whole day?
Or maybe something like 1000 cals above mantenance for the 24 hours after your workout and 1000 below for the next 24 hours untill your next session. I might try this. Anyway, report back to us about your results in what concerns muscle preservation or even gains.[b said:Quote[/b] (micmic @ May 03 2003,10:34)]Not quite, but I'm trying a similar approach. Something like eating 75% of the daily calories in the 6-7 hours after a workout. So far I like it.[b said:Quote[/b] ]Are you going to fast for a whole day?
ummmm, tell us if you hear something more specific about it, ok?[b said:Quote[/b] (Blade @ May 04 2003,4:31)]Lyle McDonald has hinted numerous times on the Keto forum that he has some strategy employing this principle, and that he would reveal it soon...so far he hasn't spilled his beans, though...![]()
One problem I can think of is this avoidance of BMR supression. I mean, if you're doing it for the health beneffits then isn't this supression supposed to play a important role in the life extension properties of caloric restriction? I've seen researchers say in a documentary on discovery chanel that most likely the two most important factors are lower body temperature and lower insulin levels and isn't lower body temperature a "side effect" of a lowered BMR?[b said:Quote[/b] (micmic @ May 04 2003,1:36)]I don't think there are many secrets to this approach, it's just concurrent bulking and cutting. Since massive feeding for upwards of 4-5 hours can upregulate leptin, the 'dieting period' of the day will be as effective as possible. Plus, you avoid BMR suppression. Plus, you better control all the factors involved in appetite regulation.
[b said:Quote[/b] ]One problem I can think of is this avoidance of BMR supression. I mean, if you're doing it for the health beneffits then isn't this supression supposed to play a important role in the life extension properties of caloric restriction?
[b said:Quote[/b] (micmic @ May 04 2003,6:11)]Sure. But with low BMR and low calories you won't build much muscle. Bodybuilding is only borderline compatible with optimal longevity practices. Longest living people around the world were undereaters, engaged in only moderate exercise and were certainly undermuscled (at least by our standards). This "i'm -doing -the -best -for -my -body" myth has fueled bodybuilders for a long time, but it's only a myth. If one's priority is life extension, he would be better off practicing yoga[b said:Quote[/b] ]One problem I can think of is this avoidance of BMR supression. I mean, if you're doing it for the health beneffits then isn't this supression supposed to play a important role in the life extension properties of caloric restriction?![]()
[b said:Quote[/b] ]I actually still haven't understand what they mean with "insulin signaling", which his supposed to be some kind of event triggered by insulin in fat cells that is involved in this acelerated ageing process. Do you know what exactly are they refering to?
There is increasing evidence that obesity is a risk factor for heart disease (independant of others), but if you look at the risks associated with it for diabetes, its clearly not good for you.[b said:Quote[/b] (micmic @ May 05 2003,11:10)]"There is no clear-cut evidence substantiating obesity causes poor health and reduced longevity (Gaesser, 1996; Ernsberger & Haskew, 1987).
Depends, theres been no decent long term trials with weight loss and longevity, the best thing is to not get fat in the first place. But in terms of the reference above, do they take into account why the person lost weight. If a large percentage of people lose weight because of disease, it will distort the picture.[b said:Quote[/b] ]Weight loss for overweight individuals may fail to improve health and can actually increase mortality rates (Andres, Muller & Sorkin, 1993).
Uhuh, obesity increases many things associated with heart disease (and involved in the atherosclerotic plaque), inflammatory mediators, cholesterol levels, BP, left ventrical hypertrophy, yada yada yada. They have to think of adipose as more than just an inert tissue, its an endocrine organ.[b said:Quote[/b] ]No relationship exists between body fat and degree of artheroscleritic buildup in coronary arteries (Barett-Connner, 1995; Kramer, et al., 1993).
Osteoporosis protection is mainly thru increased bodymass. But most obese people do not have sufficient bone mass to support their weight (especally in fat kids), so they break more bones. Cancer protection is a interesting one. The older studies on fat and lung cancer were thwarted becuase smokers are generally lighter, so it distorts the picture. Most of the breast cancer ones that seem to have shown no effect of obesity is case-control. So the measures are taken post-cancer, and cancer pateints have usually lost weight (obviously not in all examples, but in a fair few). Postmenopausal women are at a 2x greater risk of breast cancer if they are obese.[b said:Quote[/b] ]Greater body fat has a protective effect against osteoporosis (Felson, et al., 1993), lung cancer (Kabat & Wynder, 1992), and breast cancer (Wallace, et al., 1982).
increased risk of most diseases usually starts at just below the normal weight range, and increases steadily above that. Also depends on the disease you are looking at. Diabetes rates increase after about hte 'cut off' and once you get into the obese range, relative risks are HUGE.[b said:Quote[/b] ]The best mortality rates are those 25 to 30 percent over ideal recommended weight (Gaesser, 1996).
there was a paper out on this subject this year. A fat, fit person is about the same 'risk' as a unfit thin person. But they are all in the wake of the fit/thin person.[b said:Quote[/b] ]Fitness level is far more important than body weight as a predictor of longevity. Individuals as great as 50 pounds over the recommended weight in height/weight charts have lower mortality rates than thin people who were sedentary (Blair & Paffenbarger, 1994; Blair, et al., 1989).
old data. Depends on why the men where thin and why they were inactive.[b said:Quote[/b] ]Thin underactive men have 2.5 times the death rate than active overweight men."