Actually, now that you mention it....[b said:Quote[/b] (Blade @ Aug. 04 2003,1:27)]Is this a joke?
Actually, now that you mention it....[b said:Quote[/b] (Blade @ Aug. 04 2003,1:27)]Is this a joke?
Lose the attitude. There is a reason why you get these kind of reactions, my good man...[b said:Quote[/b] (Singleton @ Aug. 04 2003,2:09)]WTF, blade?
So your dumping more crap on my thread, eh restless?
[b said:Quote[/b] (Singleton @ Aug. 04 2003,4:31)]Blade,
1 - I don't care for your ill explained article.
2- You're replies don't give much insight
3 - you are dumping your passive aggressive crap on my thread. Now I do have an attitude and I did apologize. If you still have a problem with my attitude, email me, otherwise, piss off.
once this happens, satellite cell activity drops considerably. This is when you stop growing... I mean, you get bigger over time, but it is nothing like when you first started. So, we dicondition in order to try to "undo" this adaptive response to loading. Sure, SD isn't perfect, but it works.[b said:Quote[/b] (Bryan Haycock @ Aug. 04 2003,4:26)]In these cells, a sustained mechanical stress leads to assembly of two distinct structures: contractile elements containing actin filaments that create an opposing force and an elastic network that resists subsequent application of the external stress. As the stress is transmitted to the newly formed three-dimensional cytoskeleton, it is directed away from the plasma membrane, and the deformation of membrane elements near the site of force is now insufficient to activate Ca2+ influx. (Janmey PA. The cytoskeleton and cell signaling: component localization and mechanical coupling. Physiol Rev. 1998 Jul;78(3):763-81.)